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25 new of 98 responses total.
rcurl
response 40 of 98: Mark Unseen   Nov 6 06:33 UTC 1999

..and #37 is 1-propanol (old name: normal or n-propanol). 

There is no specific *charge* difference between the + and - "ends" of
a water molecule. However the water molecule has a dipole moment of
1.87E-18 e.s.u. (you asked...).
oddie
response 41 of 98: Mark Unseen   Nov 8 04:36 UTC 1999

You're right, I did :) Textbook descriptions usually say that water has
a slight charge on either end, without giving any quantitative description
of the actual magnitude of the difference, and I had assumed that it was
possible to describe the different distributions of charge in the units
of charge itself. I now know better...

You know, I might still be wrong about the isopropyl structure.  I think it
might actually be the case that the middle carbon of the propyl chain
has a -C-OH (methanol) group bonded to it; the reasoning behind the name
would be that either side of the symmetrical molecule "looks" like a
propanol chain. I'm not sure how to name such a compound under IUPAC
rules, however; maybe 2-methanol-propane??

 | | |
-C-C-C-
 | | | 
  -C-
   |
   OH
rcurl
response 42 of 98: Mark Unseen   Nov 8 06:24 UTC 1999

It is 2-methyl-1-propanol. It is, of course, a *butanol*, but there are so
many isomers of butanol that common names are available for only a few. 
It could also be called 1,1-dimethyl ethanol, but the naming convention
calls for the largest simply identifiable radical to be the basis of the
name. 
oddie
response 43 of 98: Mark Unseen   Nov 9 04:53 UTC 1999

Thankyou Rane, Russ, and Andrew (& anyone else whose name I've forgotten)
for your answers. Now I will have to think of some more questions. (:
srw
response 44 of 98: Mark Unseen   Nov 28 19:36 UTC 1999

Hmm. I would have guessed that there were only 4 isomers of butanol.
I was thinking of two different places for the OH on a straight butane, 
and two different places for an OH on an isobutane. That doesn't seem 
like so many to have common names, but maybe it is. 

The conventional names (I suspect) are:
1-butanol
2-butanol
2-methyl-1-propanol (our friend), and
2-methyl-2-propanol

As far as I can tell, none of these 4 have stereoisomers.
rcurl
response 45 of 98: Mark Unseen   Nov 28 22:27 UTC 1999

Four is "too many" (you know, one, two, three, infinity...). Three have
common names; normal (n-), secondary (sec-) and tertiary (t-) butyl
alcohols. "Our friend"  has no common name. 2-butanol has a stereoisomer
since the 2- carbon has four different groups attached. They are
d-2-methanol, and l-2-methanol (and of course the racemic mixture). 

srw
response 46 of 98: Mark Unseen   Nov 29 20:54 UTC 1999

Ok - I'll accept that four us too many. It sounded like more somehow.
Ah - yes I missed the 2-butanol stereoisomer. thx
orinoco
response 47 of 98: Mark Unseen   Nov 30 00:54 UTC 1999

If dehydration is a Bad Thing, then why does the body excrete more water, the
more it takes in?  Is there any survival value to this, or is it just a side
effect of how the relevant systems work, or what?
i
response 48 of 98: Mark Unseen   Nov 30 01:12 UTC 1999

Excess water is also a Bad Thing - the body's got to get rid of it.
orinoco
response 49 of 98: Mark Unseen   Nov 30 02:29 UTC 1999

Ah, okay.  
russ
response 50 of 98: Mark Unseen   Nov 30 05:48 UTC 1999

(Lots of things in the body, like nerves and muscles, depend on the
ion concentrations of the blood and other fluids being within certain
limits.  Too much water thins them out, and stuff starts malfunctioning.
This is bad, because you don't live too long if your nerves quit on you.)
rcurl
response 51 of 98: Mark Unseen   Nov 30 06:52 UTC 1999

It is nice, though, that the body goes to the trouble of absorbing extra
water and excreting it through a more convenient orifice, rather than just
not absorbing it. 

orinoco
response 52 of 98: Mark Unseen   Nov 30 18:59 UTC 1999

Nice why?
rcurl
response 53 of 98: Mark Unseen   Nov 30 19:53 UTC 1999

Think. 
orinoco
response 54 of 98: Mark Unseen   Nov 30 22:51 UTC 1999

I don't, remember?  It's in my handle. :)
russ
response 55 of 98: Mark Unseen   Dec 1 04:17 UTC 1999

Maybe if you'd go to the trouble of absorbing and conveniently excreting
your text, you wouldn't have this keyboard diarrhea. ;-)
keesan
response 56 of 98: Mark Unseen   Dec 6 15:51 UTC 1999

The human digestive system is open at both ends (mouth and anus).  If water
went in one end and were not absorbed where would it come out?  Orinoco, I
think you would enjoy a first-year biology class even if not required.
(I am afraid I don't get the joke in 55.)
orinoco
response 57 of 98: Mark Unseen   Dec 6 16:16 UTC 1999

Russ' remark made the situation clear enough, but thanks anyway keesan...
russ
response 58 of 98: Mark Unseen   Dec 7 03:32 UTC 1999

It was an attempt at humor on response 51.
oddie
response 59 of 98: Mark Unseen   Jan 28 04:53 UTC 2000

We are doing genetics in biology at the moment, and so I was wondering:
broadly speaking, how is eye color controlled in humans? Is there more
than one gene affecting it, more than one allele, or what?

We are doing sex-linked genes at the moment. Genetics is fascinating.
keesan
response 60 of 98: Mark Unseen   Feb 2 02:20 UTC 2000

Two genes for brown eyes make brown eyes.  Two genes for blue eyes make blue
eyes.  One of each gives you hazel/green/light brown eyes.  There are probably
several alleles of the brown-eye gene for different amounts of pigment.  Don't
know if more than one gene is involved.  But two blue eyed parents will not
produce a brown eyed child, though two brown-eyes can produce blue, as the
blue acts recessive.
oddie
response 61 of 98: Mark Unseen   Feb 2 04:55 UTC 2000

I'm not sure I understand--two "genes" for brown eyes make brown eyes, or
two alleles? In biology class we did one problem where the premise was
that there was one gene for eye color and the brown allele was dominant
over blue, but Mr. Stanley told me that this was a simplified view.
THis hypothesis explains why children of brown-eyed parents can be blue-eyed
(if both parents are heterozygous then both can pass down the recessive
blue allele) but says nothing about other colors.
If there is more than one version of the brown-eyed allele, perhaps there
is one "strong" version, fully dominant over blue-eyes, and a "weak" version
that is codominant with blue eyes?
I could look it up in the big blue tome at school entitled "Mendelian 
Inheritance in Man" but I suspect any references there would be a bit too
technical for me...
tpryan
response 62 of 98: Mark Unseen   Feb 3 01:47 UTC 2000

        My two hazel eyed parents had eight hazel eyed kids.
russ
response 63 of 98: Mark Unseen   Feb 3 04:44 UTC 2000

My two blue-eyed parents had one hazel-eyed kid, and two blue-eyed.
keesan
response 64 of 98: Mark Unseen   Feb 4 13:33 UTC 2000

I did a search on 'eye color' with AltaVista.  From the first source:


Reliable Answers on Eye Color by the MIT Guy 1

   By JJ Brannon
   
   [1]jjbrannon@aol.com
     _________________________________________________________________
   
I was a student of Salvador Luria [Nobel Laureate for Genetics] at MIT.
 Two brown-eyed parents can easily have a blue eyed child.
 Two completely blue-eyed parents CANNOT have a fully brown-eyed child with
 normal eye development except in certain extremely rare circumstances.
 The gene for brown/blue eyes is EYCL3 found on Chromosome 15.
 The gene for green/blue eyes is EYCL1 found on Chromosome 19.
 Brown is the result of melanin deposits in the iris.
 Green is the result of [this is debated] lipochrome deposits in the iris.
 Blue-grey [and in some albinism, pink] is due to a lack of pigment in the iris
.
 The underlayer, called the stroma, reflects light through its cells like
 a mirror's silver back. How the pigment is distributed over the iris involves
 other genes which produce flecks, rays, rings, partial diffusion or
 full diffusion. This inheritance is very complicated and the genes have not
 been well identified.
 Here are some reliable sources:
 http://www.ncbi.nlm.nih.gov/htbin-post/Omim/dispmim?227220
 http://www.ncbi.nlm.nih.gov/htbin-post/Omim/dispmim?227240
 http://www.gdb.org/gdb-bin/genera/genera/hgd/ObjectName/2662023?!sub=0

 Francis Galton -- Davenport & Davenport -- Bryn & Winge -- Lenz -- Hughs
 as discussed in
 Human Genetics, Chapter 5, by Reginald Ruggles Gate [1952]
 Heredity & Your Life, pp. 286-312, Boyd [1950]
     _________________________________________________________________
   
References

   1. mailto:jjbrannon@aol.com
   2. http://sln2.fi.edu/tfi/units/life/forums/anatomy/anatomy.html

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